Rtl1 CRISPR Knockout and Activation Products (h) | SCBT - Santa Cruz Biotechnology
Paternally expressed retrotransposon Gag-like 1 gene, RTL1, is one of the crucial elements for placental angiogenesis in horses†
Integrated transposons drive overexpression of Rtl1 . (A)... | Download Scientific Diagram
TDL RTL-1 Studio Monitors | Reverb
Retrotransposon gag-like 1 (RTL1) and the molecular evolution of self-targeting imprinted microRNAs | Biology Direct | Full Text
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Retrotransposon gag-like 1 (RTL1) and the molecular evolution of self-targeting imprinted microRNAs | Biology Direct | Full Text
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Silencing of retrotransposon-derived imprinted gene RTL1 is the main cause for postimplantational failures in mammalian cloning | PNAS
Rtl1 CRISPR Knockout and Activation Products (h) | SCBT - Santa Cruz Biotechnology
Identification of Rtl1, a Retrotransposon-Derived Imprinted Gene, as a Novel Driver of Hepatocarcinogenesis | PLOS Genetics
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Role of retrotransposon-derived imprinted gene, Rtl1, in the feto-maternal interface of mouse placenta | Nature Genetics
RTL1 Gene a Likely Culprit Behind Temple and Kagami-Ogata Syndromes｜Tokyo Medical and Dental University, National University Corporation
The role of eutherian‐specific RTL1 in the nervous system and its implications for the Kagami‐Ogata and Temple syndromes - Kitazawa - 2021 - Genes to Cells - Wiley Online Library
RNAi-Mediated Allelic trans-Interaction at the Imprinted Rtl1/Peg11 Locus - ScienceDirect
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TDL RTL-1 Studio Monitors | Reverb
The imprinting status of Rtl1 is brain region-dependent. (a) Schematic... | Download Scientific Diagram
Identification of Rtl1, a Retrotransposon-Derived Imprinted Gene, as a Novel Driver of Hepatocarcinogenesis | PLOS Genetics
Rtl1 CRISPR Knockout and Activation Products (m) | SCBT - Santa Cruz Biotechnology
Silencing of retrotransposon-derived imprinted gene RTL1 is the main cause for postimplantational failures in mammalian cloning | PNAS
Frontiers | Roles of retrovirus-derived PEG10 and PEG11/RTL1 in mammalian development and evolution and their involvement in human disease
